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■ Abbreviation / Long Form : DMC / 2,5-dimethyl-celecoxib

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Total Number of Papers: 24
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Abbreviation:   DMC  (>> Co-occurring Abbreviation)
Long Form:   2,5-dimethyl-celecoxib
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No. Year Title Co-occurring Abbreviation
2018 Celecoxib exerts protective effects in the vascular endothelium via COX-2-independent activation of AMPK-CREB-Nrf2 signalling. AMPK, COX, EC, HO-1
2018 Cytotoxic effect of 2, 5-dimethyl-celecoxib as a structural analog of celecoxib on human colorectal cancer (HT-29) cell line. CXB
2017 Imatinib and its combination with 2,5-dimethyl-celecoxibinduces apoptosis of human HT-29 colorectal cancer cells. COX-2, CRC
2016 Dimethyl celecoxib sensitizes gastric cancer cells to ABT-737 via AIF nuclear translocation. AIF, DAPI, PGE2
2015 2,5-Dimethyl-celecoxib inhibits cell cycle progression and induces apoptosis in human leukemia cells. COX-2
2014 Dipyrone & 2,5-dimethylcelecoxib suppress Th2-related chemokine production in monocyte. COX-2, LPS, MDC, NSAID, PBMCs, Th1, TNF, TNF-alpha
2014 The ER stress inducer DMC enhances TRAIL-induced apoptosis in glioblastoma. CHOP, DR, ER, GBM, GRP78
2012 Preferential killing of triple-negative breast cancer cells in vitro and in vivo when pharmacological aggravators of endoplasmic reticulum stress are combined with autophagy inhibitors. CQ, ERS, ERSA, TNBC
2011 Enhancement of photodynamic therapy by 2,5-dimethyl celecoxib, a non-cyclooxygenase-2 inhibitor analog of celecoxib. COX-2, ERS, PDT
10  2010 Antiangiogenic activities of 2,5-dimethyl-celecoxib on the tumor vasculature. COX-2, TuBEC
11  2010 Exploiting cyclooxygenase-(in)dependent properties of COX-2 inhibitors for malignant glioma therapy. COX-2, NSAIDs
12  2009 Enhanced killing of chemo-resistant breast cancer cells via controlled aggravation of ER stress. COX-2, ER, UMC
13  2008 Aggravated endoplasmic reticulum stress as a basis for enhanced glioblastoma cell killing by bortezomib in combination with celecoxib or its non-coxib analogue, 2,5-dimethyl-celecoxib. COX-2, ER
14  2008 Celecoxib analogs that lack COX-2 inhibitory function: preclinical development of novel anticancer drugs. ---
15  2008 Celecoxib transiently inhibits cellular protein synthesis. eIF2alpha, ER
16  2008 COX-2 inhibition is neither necessary nor sufficient for celecoxib to suppress tumor cell proliferation and focus formation in vitro. ERS, UMC
17  2008 Dimethylcelecoxib inhibits prostaglandin E2 production. cPGES, mPGES-1
18  2007 Calcium-activated endoplasmic reticulum stress as a major component of tumor cell death induced by 2,5-dimethyl-celecoxib, a non-coxib analogue of celecoxib. ER
19  2007 CCAAT/enhancer binding protein homologous protein-dependent death receptor 5 induction and ubiquitin/proteasome-mediated cellular FLICE-inhibitory protein down-regulation contribute to enhancement of tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis by dimethyl-celecoxib in human non small-cell lung cancer cells. c-FLIP, COX-2, NSCLC, TRAIL
20  2007 Reduced survivin expression and tumor cell survival during chronic hypoxia and further cytotoxic enhancement by the cyclooxygenase-2 inhibitor celecoxib. COX-2
21  2006 Antitumor properties of dimethyl-celecoxib, a derivative of celecoxib that does not inhibit cyclooxygenase-2: implications for glioma therapy. ---
22  2006 Downregulation of survivin expression and concomitant induction of apoptosis by celecoxib and its non-cyclooxygenase-2-inhibitory analog, dimethyl-celecoxib (DMC), in tumor cells in vitro and in vivo. COX-2
23  2005 Dimethyl-celecoxib (DMC), a derivative of celecoxib that lacks cyclooxygenase-2-inhibitory function, potently mimics the anti-tumor effects of celecoxib on Burkitt's lymphoma in vitro and in vivo. COX-2, NSAID
24  2005 Multitarget inhibition of drug-resistant multiple myeloma cell lines by dimethyl-celecoxib (DMC), a non-COX-2 inhibitory analog of celecoxib. COX-2, MAP, MM, STAT-3