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Total Number of Papers: 25
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| Abbreviation |
: LMP1 |
| Long Form |
: latent infection membrane protein 1 |
|
| No. |
Year |
Title |
Co-occurring Abbreviation |
| 1 |
2015 |
Characterization of the rapamycin-inducible EBV LMP1 activation system. |
EBV, JNK, LCL, MAPK, TNFR |
| 2 |
2014 |
Role of Ca2+/calmodulin-dependent kinase II-IRAK1 interaction in LMP1-induced NF-kappaB activation. |
CaMKII, EBV, IKKalpha, IL-1, KO, MEFs |
| 3 |
2013 |
Lupeol is one of active components in the extract of Chrysanthemum indicum Linne that inhibits LMP1-induced NF-kappaB activation. |
CIE, EBV, HFF, LCL |
| 4 |
2012 |
The extract of Chrysanthemum indicum Linne inhibits EBV LMP1-induced NF-kappaB activation and the viability of EBV-transformed lymphoblastoid cell lines. |
CIE, EBV, HFF, LCLs |
| 5 |
2011 |
Epstein-Barr virus-associated primary central nervous system lymphoma in a patient with adult T-cell leukemia / lymphoma. |
ATLL, CNS, EBER, EBNA2, EBV, HTLV-I, PCNSL |
| 6 |
2008 |
IRF7 activation by Epstein-Barr virus latent membrane protein 1 requires localization at activation sites and TRAF6, but not TRAF2 or TRAF3. |
EBV, IRF7 |
| 7 |
2007 |
LMP1 TRAFficking activates growth and survival pathways. |
CTAR1 and 2, EBV, TES1 and 2, TM, TNFR, TRADD |
| 8 |
2004 |
Epstein-Barr virus latent infection membrane protein 1 TRAF-binding site induces NIK/IKK alpha-dependent noncanonical NF-kappaB activation. |
EBV, MEFs, MIP, TNF |
| 9 |
2003 |
Epstein-Barr virus-encoded latent infection membrane protein 1 regulates the processing of p100 NF-kappaB2 to p52 via an IKKgamma/NEMO-independent signalling pathway. |
EBV, IKKs |
| 10 |
2003 |
Oncogenes, tumor suppressors and p52 NF-kappaB. |
EBV |
| 11 |
2003 |
TRAF1 is a critical regulator of JNK signaling by the TRAF-binding domain of the Epstein-Barr virus-encoded latent infection membrane protein 1 but not CD40. |
EBV, MAPK, NPC, TNF, TRADD, TRAFs |
| 12 |
2002 |
The Epstein-Barr virus latent membrane protein 1 putative Janus kinase 3 (JAK3) binding domain does not mediate JAK3 association or activation in B-lymphoma or lymphoblastoid cell lines. |
EBV, JAK3, LCLs, STAT3 |
| 13 |
2002 |
The oncogenic protein kinase Tpl-2/Cot contributes to Epstein-Barr virus-encoded latent infection membrane protein 1-induced NF-kappaB signaling downstream of TRAF2. |
--- |
| 14 |
2000 |
Thermodynamic characterization of the interaction between TRAF2 and tumor necrosis factor receptor peptides by isothermal titration calorimetry. |
EBV, ITC, TNFR, TRAFs |
| 15 |
1999 |
Epstein-barr virus transformation: involvement of latent membrane protein 1-mediated activation of NF-kappaB. |
EBV |
| 16 |
1999 |
Prevalence of Epstein-Barr virus in oral squamous cell carcinoma. |
EBER, EBV, ISH, OSCC, PCR |
| 17 |
1998 |
Epstein-Barr virus-transforming protein latent infection membrane protein 1 activates transcription factor NF-kappaB through a pathway that includes the NF-kappaB-inducing kinase and the IkappaB kinases IKKalpha and IKKbeta. |
NIK, TNFR |
| 18 |
1997 |
[Regulation of apoptosis by the latent infection membrane protein 1 of Epstein-Barr virus]. |
EBV |
| 19 |
1996 |
Frequent expression of interleukin-10 by Epstein-Barr virus-harboring tumor cells of Hodgkin's disease. |
ARL, EBV, HD, IL |
| 20 |
1996 |
Tumor necrosis factor receptor associated factor 2 is a mediator of NF-kappa B activation by latent infection membrane protein 1, the Epstein-Barr virus transforming protein. |
CT, TNFR |
| 21 |
1996 |
[Anti-apoptotic function of the Epstein-Barr virus LMP1 and BHRF1 proteins]. |
EBV |
| 22 |
1995 |
The Epstein-Barr virus transforming protein LMP1 engages signaling proteins for the tumor necrosis factor receptor family. |
EBV, LAP1, TRAF2 |
| 23 |
1994 |
A Ca2+/calmodulin-dependent protein kinase, CaM kinase-Gr, expressed after transformation of primary human B lymphocytes by Epstein-Barr virus (EBV) is induced by the EBV oncogene LMP1. |
EBV |
| 24 |
1993 |
Epstein-Barr virus latent membrane protein 1 is essential for B-lymphocyte growth transformation. |
EBV |
| 25 |
1992 |
Phenotypes of Epstein-Barr virus LMP1 deletion mutants indicate transmembrane and amino-terminal cytoplasmic domains necessary for effects in B-lymphoma cells. |
EBV |
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