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■ Abbreviation / Long Form : CKS1B / CDC28 protein kinase regulatory subunit 1B

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Total Number of Papers: 12
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Abbreviation:   CKS1B  (>> Co-occurring Abbreviation)
Long Form:   CDC28 protein kinase regulatory subunit 1B
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No. Year Title Co-occurring Abbreviation
2022 Regulation of microRNA miR-197-3p/CDC28 protein kinase regulatory subunit 1B (CKS1B) axis by Circular RNA hsa_circ_0000285 promotes glioma progression. ---
2021 ASF1B Promotes Oncogenesis in Lung Adenocarcinoma and Other Cancer Types. ASF1B, DHFR, GSEA, GTEx, IP-MS, LUAD, MSI, POLE3, TMB
2021 The Clinical Significance and Potential Molecular Mechanism of Upregulated CDC28 Protein Kinase Regulatory Subunit 1B in Osteosarcoma. AUC, IHC, OS, SMD
2020 Circular RNA NOX4 promotes the development of colorectal cancer via the microRNA‑485‑5p/CKS1B axis. CRC, miR, UTR
2020 Globular C1q Receptor (gC1qR/p32/HABP1) Suppresses the Tumor-Inhibiting Role of C1q and Promotes Tumor Proliferation in 1q21-Amplified Multiple Myeloma. BM, C1qRs, gC1qR, IGF2BP3, MM
2020 Molecular Network Profiling in Intestinal- and Diffuse-Type Gastric Cancer. CSC, CSE1L, DDX27, EMT, GC, GET4, IPA, MRGBP, RAE1, TOMM34
2019 Global Mapping of H3K4 Trimethylation (H3K4me3) and Transcriptome Analysis Reveal Genes Involved in the Response to Epidemic Diarrhea Virus Infections in Pigs. H3K4me3, IRF8, KLF4, OAS1, OAS2, PEDV
2017 CKS1BP7, a Pseudogene of CKS1B, is Co-Amplified with IGF1R in Breast Cancers. CKS1BP7, CNAs, DCIS, IGF1R, QM-FISH
2016 Loss of miR-1258 contributes to carcinogenesis and progression of liver cancer through targeting CDC28 protein kinase regulatory subunit 1B. HCC
10  2013 Overexpression of CDC28 protein kinase regulatory subunit 1B confers an independent prognostic factor in nasopharyngeal carcinoma. AJCC, NPC
11  2010 CDC28 protein kinase regulatory subunit 1B (CKS1B) expression and genetic status analysis in oral squamous cell carcinoma. CKS1, FISH, IHC, OLP, OSCC
12  2010 In CD28-costimulated human naive CD4+ T cells, I-kappaB kinase controls the expression of cell cycle regulatory proteins via interleukin-2-independent mechanisms. CDK2, IKK, IL, Skp2, TCR