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Abbreviation : AEs
Long Form : antiestrogens
No. Year Title Co-occurring Abbreviation
2016 Antiestrogens: structure-activity relationships and use in breast cancer treatment. ERalpha, SERMs
2013 Antiestrogen-binding site ligands induce autophagy in myeloma cells that proceeds through alteration of cholesterol metabolism. AEBS, MM
2012 Estradiol receptors in breast cancer cells: associated co-factors as targets for new therapeutic approaches. AIs, BC, ERalpha, GPER
2009 Aromatase inhibitors and breast cancer. AIs, ERalpha, PgR
2008 Pure antiestrogen-induced G1-arrest in myeloma cells results from the reduced kinase activity of cyclin D3/CDK6 complexes whereas apoptosis is mediated by endoplasmic reticulum-dependent caspases. DEX, MM, pRb, SERDs
2007 Preclinical modeling of endocrine response and resistance: focus on aromatase inhibitors. AIs, ERalpha, Grb-2, p-Shc, PgR, pMAPK
2006 Antiestrogenic therapies in solid cancers and multiple myeloma. ---
2005 Innovative drug delivery nanosystems improve the anti-tumor activity in vitro and in vivo of anti-estrogens in human breast cancer and multiple myeloma. 4-HT, MM, NPs, NS
2000 Regulation of estrogen receptor levels by ligand-induced release of compound(s) in MCF-7 cells. ER
10  1996 Active cell death induced by the anti-estrogens tamoxifen and ICI 164 384 in human mammary carcinoma cells (MCF-7) in culture: the role of autophagy. AV, TAM
11  1996 Estrogenic and antiestrogenic regulation of the half-life of covalently labeled estrogen receptor in MCF-7 breast cancer cells. ER
12  1992 Human estrogen receptor mutants with altered estrogen and antiestrogen ligand discrimination. Es
13  1991 The growth response of BG-1 ovarian carcinoma cells to estradiol, 4OH-tamoxifen, and tamoxifen: evidence for intrinsic antiestrogen activation. ---
14  1986 Antiestrogen action in the medial basal hypothalamus and pituitary of immature female rats: insights concerning relationships among estrogen, dopamine, and prolactin. DA, MBH
15  1981 Antiestrogens and ovine gonadotrophs: antagonism of estrogen-induced changes in gonadotropin secretions. ---