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Abbreviation : BBDR
Long Form : biologically based dose-response
No. Year Title Co-occurring Abbreviation
2020 Adverse outcome pathways, key events, and radiation risk assessment. AOP, KE, NCRP
2019 An evaluation of the USEPA Proposed Approaches for applying a biologically based dose-response model in a risk assessment for perchlorate in drinking water. MCLG, USEPA
2017 Can radiation research impact the estimation of risk? ---
2016 Estimation of iodine nutrition and thyroid function status in late-gestation pregnant women in the United States: Development and application of a population-based pregnancy model. mean
2013 Evaluation of iodide deficiency in the lactating rat and pup using a biologically based dose-response model. HPT
2013 Evaluation of perturbations in serum thyroid hormones during human pregnancy due to dietary iodide and perchlorate exposure using a biologically based dose-response model. HPT
2011 Marginal iodide deficiency and thyroid function: dose-response analysis for quantitative pharmacokinetic modeling. ID
2010 Development of a quantitative model incorporating key events in a hepatotoxic mode of action to predict tumor incidence. MOA, PD
2010 What role for biologically based dose-response models in estimating low-dose risk? ---
10  2009 Arsenic-induced carcinogenesis--oxidative stress as a possible mode of action and future research needs for more biologically based risk assessment. iAs, MOA, PBPK
11  2009 Competitive inhibition of thyroidal uptake of dietary iodide by perchlorate does not describe perturbations in rat serum total T4 and TSH. HPT, TSH
12  2008 A biologically based dose-response model for dietary iodide and the hypothalamic-pituitary-thyroid axis in the adult rat: evaluation of iodide deficiency. HPT, TSH
13  2007 Research toward the development of a biologically based dose response assessment for inorganic arsenic carcinogenicity: a progress report. PBPK
14  2005 Dose-incidence modeling: consequences of linking quantal measures of response to depletion of critical tissue targets. BED
15  2003 Molecular circuits, biological switches, and nonlinear dose-response relationships. ---
16  2002 The use of quantitative histological and molecular data for risk assessment and biologically based model development. TCDD
17  2001 Applications of mechanistic data in risk assessment: the past, present, and future. PBPK
18  2001 Mode of action and tissue dosimetry in current and future risk assessments. ---
19  2001 Toward a biologically based dose-response model for developmental toxicity of 5-fluorouracil in the rat: a mathematical construct. 5-FU, dNTP
20  2001 Toward a biologically based dose-response model for developmental toxicity of 5-fluorouracil in the rat: acquisition of experimental data. 5-FU, TS
21  2000 Dose-response and threshold-mediated mechanisms in mutagenesis: statistical models and study design. ---
22  2000 Evaluation of biologically based dose-response modeling for developmental toxicity: a workshop report. ---
23  1999 Effects from environmental Mn exposures: a review of the evidence from non-occupational exposure studies. MnB, MnW, RfC
24  1997 Recent advances in mathematical modeling of developmental abnormalities using mechanistic information. BMD, EBDR
25  1995 Biological modeling of 5-fluorouracil developmental toxicity. 5-FU, TS
26  1995 Research to strengthen the scientific basis for health risk assessment: a survey of the context and rationale for mechanistically based methods and models. PBPK